First published October 1, 2015 - More info
HIV-1 infection usually leads to systemic chronic inflammation that is associated with gut microbial translocation. The recently defined group 3 innate lymphoid cells (ILC3s) are critical for maintenance of intestinal barrier function; however, it is not clear whether and how HIV-1 infection influences the function of these cells. In this issue of the
Xiaohuan Guo, Yang-Xin Fu
Original citation: J Clin Invest. 2015;125(9):3430–3432. doi:10.1172/JCI83823.
Citation for this corrigendum: J Clin Invest. 2015;125(10):3992. doi:10.1172/JCI84529.
The description of the transfer experiment in the section “Disruption of ILC3s after HIV-1 infection” was incorrect. The correct sentence is below.
Specifically, human HSCs were transferred into experimental murine models and successfully developed with normal phenotype and function within lymphoid organs of humanized animals.
The authors regret the error.