The embryonic self-renewal factor SALL4 has been implicated in the development of human acute myeloid leukemia (AML). Transgenic mice expressing the human
Ailing Li, Youyang Yang, Chong Gao, Jiayun Lu, Ha-Won Jeong, Bee H. Liu, Ping Tang, Xiaopan Yao, Donna Neuberg, Gang Huang, Daniel G. Tenen, Li Chai
Overexpression of SALL4 induces enrichment of SALL4 binding and epigenetic activation markers at the