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Lack of prolactin receptor signaling in mice results in lactotroph proliferation and prolactinomas by dopamine-dependent and -independent mechanisms
Kathryn G. Schuff, Shane T. Hentges, Michele A. Kelly, Nadine Binart, Paul A. Kelly, P. Michael Iuvone, Sylvia L. Asa, Malcolm J. Low
Kathryn G. Schuff, Shane T. Hentges, Michele A. Kelly, Nadine Binart, Paul A. Kelly, P. Michael Iuvone, Sylvia L. Asa, Malcolm J. Low
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Article Genetics

Lack of prolactin receptor signaling in mice results in lactotroph proliferation and prolactinomas by dopamine-dependent and -independent mechanisms

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Abstract

Research Article

Authors

Kathryn G. Schuff, Shane T. Hentges, Michele A. Kelly, Nadine Binart, Paul A. Kelly, P. Michael Iuvone, Sylvia L. Asa, Malcolm J. Low

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Figure 4

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TH expression and activity. (a–i) In situ hybridization for TH mRNA; dar...
TH expression and activity. (a–i) In situ hybridization for TH mRNA; dark-field (a–c and g–i) and bright-field (d–f) microscopy. (a and d) Wild-type male mice express TH mRNA in the arcuate nucleus. TH mRNA is increased in Drd2–/– mice (b and e) and appears to be decreased, though clearly still evident, in Prlr–/– mice (c and f). Note comparable levels of TH mRNA in the ventral tegmental area and substantia nigra among the three genotypes: wild type (g), Drd2–/– (h), and Prlr–/– (i). Two or three mice per group were evaluated. (j) Hypothalamic and (k) striatal content of DOPA after NSD 1015 treatment. Striatal DOPA is significantly increased, and hypothalamic DOPA shows a similar trend in Drd2–/– mice, consistent with increased TH activity in dopamine neuronal terminals. DOPA levels did not differ between Prlr–/– and wild-type mice in either brain area. Scale bar represents 250 μm. Comparison was by ANOVA and Fisher post hoc test. *P < 0.05 compared with both other groups; n = 4–7.

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