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M M Kuntz, … , J B Innes, M E Weksler
Citation Information: J Clin Invest. 1979;63(1):151-153. https://doi.org/10.1172/JCI109270.
M M Kuntz, … , J B Innes, M E Weksler
Published January 1, 1979
Citation Information: J Clin Invest. 1979;63(1):151-153. https://doi.org/10.1172/JCI109270.
Abstract
The autologous mixed lymphocyte reaction is impaired in patients with systemic lupus erythematosus. This is because nonthymus-derived (T)-lymphocyte preparations from such patients do not stimulate autologous T-lymphocyte proliferation normally. This defect may explain the impaired generation of suppressor activity in systemic lupus erythematosus and thereby the occurrence of autoantibodies in this disease.
Authors
M M Kuntz, J B Innes, M E Weksler
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