Abstract
A quantitative primate model of arterial thromboembolism has been characterized with respect to mechanism and usefulness in evaluating modifying variables. The model involved the kinetic measurements of 51Cr-platelets and 125I-fibrinogen consumption by femoral arteriovenous cannulae in chaired baboons.
Authors
Laurence A. Harker, Stephen R. Hanson, Thomas R. Kirkman
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