Article tools
  • View PDF
  • Cite this article
  • E-mail this article
  • Send a letter
  • Information on reuse
  • Standard abbreviations
  • Article usage
Author information
Need help?


Impaired autophagic flux mediates acinar cell vacuole formation and trypsinogen activation in rodent models of acute pancreatitis

Olga A. Mareninova, Kip Hermann, Samuel W. French, Mark S. O’Konski, Stephen J. Pandol, Paul Webster, Ann H. Erickson, Nobuhiko Katunuma, Fred S. Gorelick, Ilya Gukovsky and Anna S. Gukovskaya

Published April 1, 2013

Original citation: J. Clin. Invest. 2009;119(11):3340–3355. doi:10.1172/JCI38674.

Citation for this corrigendum: J. Clin. Invest. 2013;123(4):1402. doi:10.1172/JCI69660.

For Figure 2A, the authors did not indicate that the two lanes were noncontiguous.

(A) LC3-I to LC3-II conversion (immunoblot) in pancreas of rats under conditions of fasting (for 17 hours) and pancreatitis (see Methods). ERK1/2 served as loading control. Lanes were run on the same gel but were noncontiguous.

The authors regret the error.