Graft rejection remains a formidable problem contributing to poor outcomes after lung
transplantation. Blocking chemokine pathways have yielded promising results in some
organ transplant systems. Previous clinical studies have demonstrated upregulation of
CCR2 ligands following lung transplantation. Moreover, lung injury is attenuated in CCR2‐
deficient mice in several inflammatory models. In this study, we examined the role of CCR2
in monocyte recruitment and alloimmune responses in a mouse model of vascularized …