Cell proliferation and fiber formation in chronic carbon tetrachloride intoxication: A morphologic and chemical study
E Rubin, F Hutterer, H Popper - The American Journal of …, 1963 - ncbi.nlm.nih.gov
E Rubin, F Hutterer, H Popper
The American Journal of Pathology, 1963•ncbi.nlm.nih.govNecrosis. After io days of treatment, centrilobular necrosis and hydropic degeneration of
hepatocytes had developed, with accumulation of mesenchymal cells and moderate central
and midzonal steatosis. Reticulin and aniline blue stained fibers were increased in the
necrotic central zones (Fig. i). Many labeled hepatocytes were evident (Fig. 2). Labeling of
mesenchymal cells had also increased, butnot as muchas had
hepatocytes had developed, with accumulation of mesenchymal cells and moderate central
and midzonal steatosis. Reticulin and aniline blue stained fibers were increased in the
necrotic central zones (Fig. i). Many labeled hepatocytes were evident (Fig. 2). Labeling of
mesenchymal cells had also increased, butnot as muchas had
Necrosis. After io days of treatment, centrilobular necrosis and hydropic degeneration of hepatocytes had developed, with accumulation of mesenchymal cells and moderate central and midzonal steatosis. Reticulin and aniline blue stained fibers were increased in the necrotic central zones (Fig. i). Many labeled hepatocytes were evident (Fig. 2). Labeling of mesenchymal cells had also increased, butnot as muchas had
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