Publisher Summary The human body is equipped with mechanisms for the provision and
maintenance of tissue cholesterol levels. Cholesterol absorption increases with dietary
intake, with no upper limit. In times of reduced intake, the liver and small intestine increase
their synthesis of cholesterol. Absorbed cholesterol is transported to the liver as chylomicron
remnants and hepatic cholesterol is either secreted as “very low-density lipoproteins,” which
are catabolized in part to low-density lipoproteins (LDL), or secreted into bile as free …