Since the identification of cholesteryl ester transfer protein (CETP), its role in the modulation
of HDL levels and cardiovascular disease has been debated. With the early detection of
genetic variants followed by the finding of families deficient in CETP, genetic studies have
played a large role in the attempts to understand the association of CETP with lipids and
disease; however, results of these studies have often led to disparate conclusions. With the
availability of a greater variety of genetic polymorphisms and larger studies in which disease …