Endocrinopathies in the family of endoplasmic reticulum (ER) storage diseases: disorders of protein trafficking and the role of ER molecular chaperones
PS Kim, P Arvan - Endocrine reviews, 1998 - academic.oup.com
PS Kim, P Arvan
Endocrine reviews, 1998•academic.oup.comA. Escape from ER retention as one hypothesis to explain anterograde protein traffic from
the ER B. Cargo receptors as another hypothesis to explain anterograde protein traffic from
the ER C. What provides quality control of ER export? D. ER-associated degradation IV.
Endocrinopathies as Models of Defective Protein Export A. Congenital hypothyroid goiter
with thyroglobulin deficiency
the ER B. Cargo receptors as another hypothesis to explain anterograde protein traffic from
the ER C. What provides quality control of ER export? D. ER-associated degradation IV.
Endocrinopathies as Models of Defective Protein Export A. Congenital hypothyroid goiter
with thyroglobulin deficiency
A. Escape from ER retention as one hypothesis to explain anterograde protein traffic from the ER B. Cargo receptors as another hypothesis to explain anterograde protein traffic from the ER C. What provides quality control of ER export? D. ER-associated degradation IV. Endocrinopathies as Models of Defective Protein Export A. Congenital hypothyroid goiter with thyroglobulin deficiency
Oxford University Press