Left ventricular remodeling after myocardial infarction: pathophysiology and therapy
MGSJ Sutton, N Sharpe - Circulation, 2000 - Am Heart Assoc
MGSJ Sutton, N Sharpe
Circulation, 2000•Am Heart Assocrenin-angiotensin-aldosterone system, and stimulates the production of atrial and brain
natriuretic peptides (ANP and BNP). 10 Augmented shortening and increased heart rate
from sympathetic stimulation result in hyperkinesis of the noninfarcted myocardium and
temporary circulatory compensation. In addition, the natriuretic peptides reduce
intravascular volume and systemic vascular resistance, normalize ventricular filling, and
improve pump function.
natriuretic peptides (ANP and BNP). 10 Augmented shortening and increased heart rate
from sympathetic stimulation result in hyperkinesis of the noninfarcted myocardium and
temporary circulatory compensation. In addition, the natriuretic peptides reduce
intravascular volume and systemic vascular resistance, normalize ventricular filling, and
improve pump function.
renin-angiotensin-aldosterone system, and stimulates the production of atrial and brain natriuretic peptides (ANP and BNP). 10 Augmented shortening and increased heart rate from sympathetic stimulation result in hyperkinesis of the noninfarcted myocardium and temporary circulatory compensation. In addition, the natriuretic peptides reduce intravascular volume and systemic vascular resistance, normalize ventricular filling, and improve pump function.
Am Heart Assoc