Crucial role of the protein C pathway in governing microvascular inflammation in inflammatory bowel disease J. Clin. Invest.
117:7
doi:10.1172/JCI31027
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Figure 7 Inhibition of T cell adhesion to TNF-α–inflamed HIMECs by recombinant aPC.
HIMEC monolayers were left untreated or stimulated with TNF-α with or without recombinant human aPC. Calcein-labeled MOLT4 T cells were added to the HIMEC monolayers. Values are mean ± SEM of 4 separate experiments. *P < 0.05 versus TNF-α without aPC.
