Roger S.-Y. Foo, Kartik Mani, Richard N. Kitsis
J Clin Invest.
2005;
115(3):565–571
doi:10.1172/JCI24569
This article Copyright © 2005, The American Society for Clinical Investigation
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R
ecently, low — but abnormal — rates of cardiomyocyte apoptosis have been observed in failing human hearts. Genetic and pharmacological studies suggest that this cell death is causally linked to heart failure in rodent models. Herein, we review these data and discuss potential therapeutic implications.
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