Dysbindin-1 is reduced in intrinsic, glutamatergic terminals of the hippocampal formation in schizophrenia
J. Clin. Invest. Konrad Talbot, et al. 113:1353 doi:10.1172/JCI20425 [Go to this article.]

Figure 1
Dysbindin gene expression in normal human HF, demonstrated by in situ hybridization. The tissue was counterstained for Nissl substance. All principal neurons in the DG and CA1_3 were labeled with the antisense riboprobe, as seen at low magnification (A). Gene expression in CA1 (B) and in the DGg (E) is lower than in CA3 (C) or in the DGh (D). Comparison of DGg with antisense (E) and with sense riboprobes (F) shows that the latter produced very little cell labeling. Scale bar in A: 1 mm; scale bars in B_F: 20 ∝m.