Impaired glucose phosphorylation and transport in skeletal muscle cause insulin resistance in HIV-1–infected patients with lipodystrophy
J. Clin. Invest. Georg M.N. Behrens, et al. 110:1319 doi:10.1172/JCI15626 [Go to this article.]

Figure 5
Differences in the distribution volume (DVCE) and the PF of F-18-FDG metabolism. DVCE (left panel) reflects glucose transport, and PF (right panel) quantitatively describes the relative influence of glucose phosphorylation on the overall rate of glucose utilization. Both parameters were significantly higher in therapy-naive patients (white regions) than in patients with lipodystrophy on HAART (gray regions).
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