JCI - Impaired glucose phosphorylation and transport in skeletal muscle cause insulin resistance in HIV-1

Impaired glucose phosphorylation and transport in skeletal muscle cause insulin resistance in HIV-1–infected patients with lipodystrophy
J. Clin. Invest. 110:9 doi:10.1172/JCI15626 [Go to this article.]

Options: View larger image (or click on image)

Figure 1

Three-compartment model for the F-18-FDG kinetics of the PET data. Dynamically acquired PET data and arterial plasma time course of F-18-FDG activity were used as input functions in the compartmental modeling. The rate constants represent inward transport (k₁), outward transport (k₂), and phosphorylation (k₃) of F-18-FDG.