A A Rossini, J P Mordes, D L Greiner, K Nakano, M C Appel, E S Handler
J Clin Invest.
1986;
77(4):1399–1401
doi:10.1172/JCI112448
This article Copyright © 1986, The American Society for Clinical Investigation
Abstract
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e report that transfusions of RT1u Wistar-Furth (WF) spleen cells prevented spontaneous diabetes in the RT1u BB/W rat while RT1b Buffalo rat spleen cells did not. In addition, donor origin WF T lymphocytes were detected in nondiabetic-susceptible BB/W recipients 5 mo after transfusion. Survival of donor-origin lymphocytes may provide the cellular mechanism by which major histocompatibility complex-compatible WF spleen cell transfusions prevent BB rat diabetes.
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